Lactate induces vascular permeability via disruption of VE-cadherin in endothelial cells during sepsis

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Lactate induces vascular permeability via disruption of VE-cadherin in  endothelial cells during sepsis
Protein Interactions at Endothelial Junctions and Signaling
Lactate induces vascular permeability via disruption of VE-cadherin in  endothelial cells during sepsis
Lactate promotes macrophage HMGB1 lactylation, acetylation, and
Lactate induces vascular permeability via disruption of VE-cadherin in  endothelial cells during sepsis
Interleukin-1RA Mitigates SARS-CoV-2–Induced Inflammatory Lung
Lactate induces vascular permeability via disruption of VE-cadherin in  endothelial cells during sepsis
Lactate induces vascular permeability via disruption of VE
Lactate induces vascular permeability via disruption of VE-cadherin in  endothelial cells during sepsis
Lactate promotes macrophage HMGB1 lactylation, acetylation, and
Lactate induces vascular permeability via disruption of VE-cadherin in  endothelial cells during sepsis
Oxidative Stress and Endothelial Dysfunction in Sepsis and Acute
Lactate induces vascular permeability via disruption of VE-cadherin in  endothelial cells during sepsis
Diagnosing capillary leak in critically ill patients: development
Lactate induces vascular permeability via disruption of VE-cadherin in  endothelial cells during sepsis
Lactate released by inflammatory bone marrow neutrophils induces
Lactate induces vascular permeability via disruption of VE-cadherin in  endothelial cells during sepsis
The distribution of VE-cadherin in cultured endothelial cells
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